Category 1
3/5

MOTS-C.

Mitochondrial-derived peptide that enhances metabolic flexibility, glucose uptake, and exercise capacity through AMPK activation.

MetabolicFat LossExercise PerformanceMitochondrial
AMPK Activator Directly activates AMPK signaling, mimicking the metabolic benefits of exercise at the cellular level Lee et al., Cell Metabolism, 2015

Quick Reference.

Also Known As Mitochondrial ORF of the 12S rRNA Type-C
Class Mitochondrial-Derived Peptide (MDP)
Molecular Weight 2,174.6 Da (16 amino acids)
Administration Subcutaneous injection
Half-Life Estimated 4-8 hours (limited pharmacokinetic data)
Legal Status Research compound — FDA Category 1
Encoded By Mitochondrial DNA (12S rRNA gene)
◆ ◆ ◆

Mechanism of Action.

MOTS-c is a 16-amino-acid peptide encoded by the mitochondrial genome within the 12S rRNA gene, making it one of several mitochondrial-derived peptides (MDPs) with signaling functions. Its primary mechanism involves activation of the AMPK (AMP-activated protein kinase) pathway, a master metabolic sensor. MOTS-c inhibits the folate-methionine cycle, leading to accumulation of AICAR (5-aminoimidazole-4-carboxamide ribonucleotide), which activates AMPK. This triggers downstream effects including enhanced glucose uptake via GLUT4 translocation, increased fatty acid oxidation, improved mitochondrial biogenesis, and suppression of the mTOR pathway. MOTS-c also translocates to the nucleus during metabolic stress, directly regulating gene expression through interactions with ARE (antioxidant response element) and NRF2 pathways.

Research Summary.

The foundational study by Lee et al. (2015) in Cell Metabolism demonstrated that MOTS-c administration prevented diet-induced obesity and insulin resistance in mice. Subsequent research showed that MOTS-c levels decline with age, correlating with metabolic dysfunction. A 2019 study found that circulating MOTS-c levels increase with exercise in humans, suggesting it functions as an exercise-mimetic signal. Research has shown MOTS-c improves glucose homeostasis in aged mice, enhances exercise capacity, and may protect against age-related metabolic decline. Studies also indicate anti-inflammatory properties and potential cardiovascular benefits through endothelial function improvement.

Side Effects & Safety.

MOTS-c is an endogenous peptide that the body naturally produces, which theoretically supports a favorable safety profile. However, as a research compound, there are no completed human clinical trials establishing formal safety data. Preclinical studies have not identified significant adverse effects at physiological doses. Theoretical concerns include potential interactions with other AMPK-modulating compounds and unknown effects of supraphysiological dosing on mitochondrial dynamics and cellular energy balance. The relatively recent discovery of MOTS-c (2015) means long-term research data is limited.

Legal Status & Access.

MOTS-c is classified as a Category 1 research peptide. It is not FDA-approved for any clinical indication and has not yet entered formal clinical trials. It is available from research peptide suppliers for laboratory investigation.
◆ ◆ ◆

Where to Source MOTS-C.

Verified research peptide suppliers

BioPure Peptides

Premium research peptides with third-party COAs. Use code POWER at checkout.

Code: POWER
Shop Now →

Midwest Peptide

US-based peptide supplier. 10% off with code POWER, 30-day cookie.

Code: POWER
Shop Now →

Offline Peptides

75+ peptides, HPLC-verified, third-party COAs. Free shipping $250+.

Shop Now →
◆ ◆ ◆

Frequently Asked Questions.

Related Peptides.

Category 1

AOD-9604

3/5
Research

5-Amino-1MQ

3/5
Investigational

SS-31

3/5
Research

NAD+

3/5

Research Disclaimer: Content on PowerPeptides.co is for informational and research purposes only. It is not medical advice. Peptides discussed are research compounds unless explicitly noted as FDA-approved. Always consult a licensed healthcare provider before beginning any peptide protocol. Full Disclaimer | Affiliate Disclosure